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Resverlogix Corp T.RVX

Alternate Symbol(s):  RVXCF

Resverlogix Corp. is a Canada-based late-stage biotechnology company. The Company is engaged in epigenetics, with a focus on developing therapies for the benefit of patients with chronic diseases. Its epigenetic therapies are designed to regulate the expression of disease-causing genes. The Company's clinical program is focused on evaluating its lead candidate apabetalone (RVX-208) for the treatment of cardiovascular disease and associated comorbidities, and post-COVID-19 conditions. RVX-208 is a small molecule that is a selective bromodomain and extra-terminal (BET) inhibitor. BET bromodomain inhibition is an epigenetic mechanism that can regulate disease-causing genes. RVX-208 is a BET inhibitor selective for the second bromodomain (BD2) within the BET proteins. It partners with EVERSANA, to support the commercialization of RVX-208 for cardiovascular disease, post-COVID-19 conditions, and pulmonary arterial hypertension in Canada and the United States.


TSX:RVX - Post by User

Bullboard Posts
Comment by BearDownAZon Jan 25, 2016 9:57pm
159 Views
Post# 24492634

RE:RE:RE:INTERESTING ARTICLE FROM CLEVELAND CLINIC

RE:RE:RE:INTERESTING ARTICLE FROM CLEVELAND CLINICInteresting findings about the paradoxical relationship of plaque calicification and plaque stabilization. I don't have anything to add to this concise, well-written article.

This passage from the article pretty much sums it up:
"The increase in calcification revealed by the analysis may represent a means by which statin therapy stabilizes coronary plaque to reduce the risk of cardiovascular events, says the study’s senior investigator, Steven Nissen, MD, who adds that this hypothesis remains to be proved.
 
“We found that as plaques were getting smaller with statins, they were calcifying,” explains Dr. Nissen, Chairman of the Robert and Suzanne Tomsich Department of Cardiovascular Medicine at Cleveland Clinic.
 
“It’s exactly the opposite of what you might think intuitively,” he continues. “This is an important observation that tells us that statins work to stabilize plaques by converting softer, cholesterol-laden plaques that are prone to rupture into more stable calcified plaques that are relatively inert. It explains the paradox of why serial measurement of calcium doesn’t necessarily work to track the progression of disease, and it explains to some extent how statins work.”" 
Bullboard Posts