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Theratechnologies Inc T.TH

Alternate Symbol(s):  THTX

Theratechnologies Inc. is a Canada-based clinical-stage biopharmaceutical company. The Company is focused on the development and commercialization of therapies addressing unmet medical needs. It markets prescription products for people with human immunodeficiency viruses (HIV) in the United States. The Company's research pipeline focuses on specialized therapies addressing unmet medical needs in HIV, nonalcoholic steatohepatitis (NASH) and oncology. Its medicines include Trogarzo and EGRIFTA SV (tesamorelin for injection). Trogarzo (ibalizumab-uiyk) injection is a long-acting monoclonal antibody which binds to domain 2 of the CD4 T cell receptors. EGRIFTA SV (tesamorelin for injection) is approved in the United States for the reduction of excess abdominal fat in people with HIV who have lipodystrophy. Its portfolio includes Phase I clinical trial of sudocetaxel zendusortide (TH1902), a novel peptide-drug conjugate (PDC), in patients with advanced ovarian cancer.


TSX:TH - Post by User

Comment by qwerty22on Jul 23, 2021 12:58pm
117 Views
Post# 33596513

RE:RE:RE:RE:RE:RE:RE:RE:RE:RE:RE:RE:RE:RE:RE:TH2101

RE:RE:RE:RE:RE:RE:RE:RE:RE:RE:RE:RE:RE:RE:RE:TH2101

"I do recall in the new presentation their new diagram specifically shows that the Sortilin is recycled and spit out the cell again to attach to the membrane.  That was the first time I saw that.  No doubt they understand the amount and timing of recycle are important factors to  learn in trials."

That would be a big presumption to make. It would take a lot of science to get to that level of understanding. It seems much more likely to me they are just taking the general model of endosomal processing as it's understood in the scientific literature and applying it to their situation. Similar to the NASH schematic for the affect of GH/IGF-1. The company hasn't proved all those processes occur when you dose with Egrifta, they've just taken the general model of the GH/IGF-1 axis impact on the liver from the scientific literature.

If they could do what you say then the likelihood is they'd already have a much deeper understanding of the MOA of the drug than they seem to.


Wino115 wrote:

Thank you for digging in.  I do recall in the new presentation their new diagram specifically shows that the Sortilin is recycled and spit out the cell again to attach to the membrane.  That was the first time I saw that.  No doubt they understand the amount and timing of recycle are important factors to  learn in trials.   Very helpful and I think you found the UnivGotenburg papers that describe what they are aiming to do, which is very different from THTX.  As you point out, they are specifically looking for an anti-Sortilin therapy and really focusing on stopping metasticism function more than anything else. 

The Feb 2021 UGoth paper states:  "Data suggest that co-expression of progranulin and its receptor sortilin is a novel prognostic biomarker combination identifying a highly malignant subgroup of breast cancer. Importantly, this subpopulation could potentially be targeted with anti-sortilin based therapy"

I also see they focus on IL6 which I have no clue what it is, but wonder if we will hear about that in the future.  


" Sortilin is apart from being a receptor to progranulin also described to bind with high-affinity to the proinflammatory cytokines IL6. In breast cancer, IL6 is associated with increased tumor stage, lymph node infiltration, recurrence, and treatment resistance further signifying the role of sortilin in breast cancer progression.

Thanks for helping us get the Sortilin / stem cell connection. All good stuff and glad we are way ahead of them in moving to trials. 

 

jfm1330 wrote: I am not a cancer expert at all, but I know enough about biochemistry to understand some stuff, to a certain extent, when I do searches about topics of interest and when I read relevant scientific articles. Biochemistry is like a language, when you master enough of that language, it allows you to learn more of that language. That's what I try to do here, use my relatively basic knowledge in biochemistry to try to discover new elements and understand them enough to be able to explain here the basic principles behind them. Again. I am not sure I am always well understood by all, but at least I try to make it as accessible as possible to non science people. And by the way, rest assure that Marsolais, Beliveau and others at Thera know much more than me, it's not even close. I am sure they would not learn anything from me, even though I still wonder why they are not even hinting at radioisotopes as an imaging tool and a possible therapeutic avenue in future PDCs...

 

 

realitycheck4u wrote:

You are such an incredible wealth of information and knowledge that I cannot see how or why you have retired. You truly could possible help in untold ways with cancer patients.  Would you like an introduction to Paul and possible lend an hour or two each week to the team at Thera?  I'm certain I (or we) can make that happen.  Let me know privately or here.

 

jfm1330 wrote: I am unable to find a website for Sortina Pharma. That being said searching with some key words I found some interesting scientific articles. I found the following about the role of sortilin in breast cancer:

Interestingly, sortilin has been associated with metastatic potential in breast cancer [17] and is highly expressed in breast cancer cell lines compared to non-tumorigenic breast epithelial cells [17]. In addition, siRNA knockdown of sortilin inhibited breast cancer cell adhesion, migration and invasion suggesting that sortilin indeed is involved in breast cancer progression 

https://breast-cancer-research.biomedcentral.com/articles/10.1186/s13058-018-1060-5

So if you elimiate the expression of sortilin with siRNA, you end up suppressing signs of cancer aggressivity. What needs to be understood is that by eliminating sortilin from the cancer cell membrane, you eliminate the receptor for progranulin, so you deregulate the axis sortilin-progranulin. But when you think about it, a sufficient enough dose of TH1902 is doing somenthing similar to siRNA. TH1902 by saturating sortilin ends up having it out of the cell membrane and all internalized inside the cell. So TH1902, like siRNA, is also messing up the sortilin-progranulin axis involved in cancer aggressivity. So all that is probably linked to the inhibition of the vasculomimicry process that is involved in cancer aggressivity.

From the little I have been able to find, Sortina approach is to block sortilin capacity to bind progranulin with a small molecule, and that would also be a way to mess up the sortilin-progranulin axis. This would be in line with my suggestion to use TH19P01 alone as anti-cancer agent complementary to PDCs based on it. But for that to work you would need a long acting release formulation like the one that exists for octreotide (Sandostatin LAR) in the treatment of neuroendocrine tumors, or a more stable analog of TH19P01. In this case the peptide octreotide (analog of the natural peptide somatostatin), is also the peptidic part of the PDC Lu177-Dotatate (Lutathera). So to treat neuroendorine cancers, they use octreotide LAR for chronic treatment, and the PDC based on octreotide (Lu177-Dotatate) for acute treatment. A similar thing, in theory, would be possible with TH19P01 and PDCs based on TH19P01, like TH1902 or TH2101.

I am talking about this just because it seems clear to me that sortilin could be used in different ways to fight cancer, and a similar combo therapy already exist to treat neuroendocrine tumors, with Sandostatin LAR and Lutathera. I know it's still a bit science fiction at the moment, but not totally. The path is clear, it just needs to be explored. But if TH1902, Thera will be able to gather the necessary ressouces to explore many possibilities related to sortilin.


 

 

Wino115 wrote:

If we get POC, the hard part will be ranking which approach to do next, and then next, and then next!

On the CSC and Sortilin question, I recall that the Univ of Gothenburg team and their spinoff venture (Sortina Pharma) is studying the Sortilin/stem cell nexus for their mTNBC drug they hope to figure out.  Their website or Univ research may have more on it. 

 


 

 


 




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