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Theralase Technologies Inc. V.TLT

Alternate Symbol(s):  TLTFF

Theralase Technologies Inc. is a Canada-based clinical-stage pharmaceutical company. The Company is engaged in the research and development of light activated compounds and their associated drug formulations. The Company operates through two divisions: Anti-Cancer Therapy (ACT) and Cool Laser Therapy (CLT). The Anti-Cancer Therapy division develops patented, and patent pending drugs, called Photo Dynamic Compounds (PDCs) and activates them with patent pending laser technology to destroy specifically targeted cancers, bacteria and viruses. The CLT division is responsible for the Company’s medical laser business. The Cool Laser Therapy division designs, develops, manufactures and markets super-pulsed laser technology indicated for the healing of chronic knee pain. The technology has been used off-label for healing numerous nerve, muscle and joint conditions. The Company develops products both internally and using the assistance of specialist external resources.


TSXV:TLT - Post by User

Comment by CancerSlayeron Mar 03, 2022 3:07am
697 Views
Post# 34477884

RE:New Australian Study Discusses TLD1433

RE:New Australian Study Discusses TLD1433

Loooong day at work...thanks Eoganacht for your continuing enlightenment of our ACT's potential.  Dr. Mc Farland's successful research into the medicinal benefits of organometallic chemistry has truly opened the floodgates for future research & clinical trials re: this miraculous compound/technology, & it's nice to know TLT will ultimately be the beneficiary of its success.  

What I find very encouraging is not only the recent wealth of research now being done in this field (in large part due to Dr. Mc Farland), but also the high degree of versatility this compound possesses   outside of its ACT potential (I.e. the myriad formulations, treatment types & medical applications)...this unique attribute sometimes gets understated.  I am particularly looking forward to clinical studies of its potential use as a combo or adjuvant drug.  This is another area of need that TLT can address.  

There are many combo treatment studies using Ruthenium complexes in cancer treatment that show great potential in offering synergistic advantages as well as the potential for reducing drug dosage (of TLTD-1433 &/or its partner drug), treatment toxicity & drug resistance.  This type of clinical use could conceivably generate more revs than if used as a stand-alone option...possibly more so in the short term.  Its use in combination with more conventional therapies could have the benefit of easing its clinical acceptance/adoption....helping transition docs out of their comfort zone of long-standing/more accepted practices to newer & less familiar protocols.

The following is just another example (abstract only) showing the promise & versatility of organometallic medicine/Ru complexes in defeating cancer...published today (3/2/2022).  The study highlights the immunotherapeutic potential of Ru complexes that demonstrate the ability to reduce the effects of Tumor Associated Macrophages (TAMS)...TAMS exert pro-tumor effects by suppressing the body's natural immune response to cancer.
GLTA..

Metalloimmunotherapy with rhodium and ruthenium complexes: Targeting tumor-associated macrophages

 

Abstract

Tumor associated macrophages (TAMs) suppress the cancer immune response and are a key target for immunotherapy. The effects of ruthenium and rhodium complexes on TAMs have not been well characterized. To address this gap in the field, a panel of 22 dirhodium and ruthenium complexes were screened against three subtypes of macrophages, triple-negative breast cancer and normal breast tissue cells. Experiments were carried out in 2D and biomimetic 3D co-culture experiments with and without irradiation with blue light. Leads were identified with cell-type-specific toxicity toward macrophage subtypes, cancer cells, or both. Experiments with 3D spheroids revealed complexes that sensitized the tumor models to the chemotherapeutic doxorubicin. Cell surface exposure of calreticulin, a known facilitator of immunogenic cell death (ICD), was increased upon treatment, along with a concomitant reduction in the M2-subtype classifier arginase. Our findings lay a strong foundation for the future development of ruthenium- and rhodium-based chemotherapies targeting TAMs.


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