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Oncolytics Biotech Inc T.ONC

Alternate Symbol(s):  ONCY

Oncolytics Biotech Inc. is a clinical-stage biotechnology company. The Company is focused on developing pelareorep, an intravenously delivered immunotherapeutic agent that activates the innate and adaptive immune systems and weakens tumor defense mechanisms. This compound induces anti-cancer immune responses and promotes an inflamed tumor phenotype turning cold tumors hot through innate and adaptive immune responses to treat a variety of cancers. This improves the ability of the immune system to fight cancer, making tumors more susceptible to a broad range of oncology treatments. The Company’s primary focus is to advance its programs in hormone receptor-positive / human epidermal growth factor 2- negative (HR+/HER2-) metastatic breast cancer and advanced/metastatic pancreatic ductal adenocarcinoma to registration-enabling clinical studies. In addition, it is exploring opportunities for registrational programs in other gastrointestinal cancers through its GOBLET platform study.


TSX:ONC - Post by User

Post by Noteableon Sep 03, 2022 12:26pm
373 Views
Post# 34940081

May 2022-ESMO - pelareorep "primes" the TME for ICI -AWARE-1

May 2022-ESMO - pelareorep "primes" the TME for ICI -AWARE-1

The oncolytic virus pelareorep primes the tumor microenvironment for checkpoint blockade therapy in early breast cancer patients -results from AWARE-1 study


Background

Pelareorep (pela) is an intravenously delivered non-engineered oncolytic reovirus demonstrating anti-tumor activity through innate and adaptive immune responses. Previous data from the AWARE-1 study demonstrated that addition of atezolizumab (atezo) to pela increased the CelTIL score (study’s primary endpoint) by more than 30% in 60% of HR+/HER2- early breast cancer (BC) patients.

CelTIL is a composite measure of tumor cellularity and tumor-infiltrating lymphocytes (TILs) and has been associated with a better prognosis in BC. The increase in CelTIL score observed in AWARE-1 was accompanied by PD-L1 upregulation.

 

Results

IHC showed a significant increase (p-value=0.04) in caspase 3 staining in all patients from day3 to day 21 that reached significance in C2. PAM50 analysis indicated a conversion of baseline disease from luminal B to luminal A in both cohorts, with 100% of patients converting to luminal A in C2. TCR-seq showed a decrease in pre- vs. post-treatment blood T cell diversity in both cohorts, which was significant (p-value=0.01) in C2. An association between decreased post-treatment T cell diversity and pre- vs. post-treatment increases in TILs was observed in both cohorts, reaching significance in C2 (p-value=0.01). DSP showed an increase in activated T cells but not exhausted T cells in both cohorts.

Conclusions

These data show that pela induces an inflamed tumor phenotype and demonstrate its synergy with atezo. Moreover, these data support pela’s immune-based mechanism of action and suggest that combining pela with atezo may improve outcomes in BC.


https://oncologypro.esmo.org/meeting-resources/esmo-breast-cancer-congress/the-oncolytic-virus-pelareorep-primes-the-tumor-microenvironment-for-checkpoint-blockade-therapy-in-early-breast-cancer-patients-results-from-awar

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