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Theralase Technologies Inc. V.TLT

Alternate Symbol(s):  TLTFF

Theralase Technologies Inc. is a Canada-based clinical-stage pharmaceutical company. The Company is engaged in the research and development of light activated compounds and their associated drug formulations. The Company operates through two divisions: Anti-Cancer Therapy (ACT) and Cool Laser Therapy (CLT). The Anti-Cancer Therapy division develops patented, and patent pending drugs, called Photo Dynamic Compounds (PDCs) and activates them with patent pending laser technology to destroy specifically targeted cancers, bacteria and viruses. The CLT division is responsible for the Company’s medical laser business. The Cool Laser Therapy division designs, develops, manufactures and markets super-pulsed laser technology indicated for the healing of chronic knee pain. The technology has been used off-label for healing numerous nerve, muscle and joint conditions. The Company develops products both internally and using the assistance of specialist external resources.


TSXV:TLT - Post by User

Post by Eoganachton Nov 14, 2024 12:08pm
186 Views
Post# 36312953

Sonodyhamic Therapy Study Cites Advantages of Rutherrin

Sonodyhamic Therapy Study Cites Advantages of RutherrinThe following study of Sonodynamic Therapy notes how PDT with Rutherrin makes brain tumours vulnerable to immunological treatments. The use of Rutherrin PDT could be a turning point in treating hard-to-treat GBM, both as a monotherapy and/or a combo treatment which gives established treatments a fighting chance of working.

Hopefully a Phase 1 Clinical trial of Rutherrin PDT for GBM will begin in 2025. 

The abscopal effects of sonodynamic therapy in cancer

Victoria G. Collins, Dana Hutton, Kismet Hossain-Ibrahim, James Joseph & Sourav Banerjee
 
British Journal of Cancer (2024)
 
Conversion of immunologically cold tumours
 
Glioblastoma multiforme (GBM) has an immunologically “cold” TME created by a lack of strong TAAs, secretion of immunosuppressive factors, and occasionally downregulated MHCs [73,74,75]. This is defined by the degree of T cell infiltration. In comparison, a “hot” tumour is T cell-infiltrated and susceptible to T cell-mediated attack [76, 77]. However, GBMs also have an immunosuppressed myeloid landscape that works to control immune response while allowing tumour cell proliferation [78,79,80,81]. These mechanisms mean that immunotherapy has little effect on GBM [82].
 
One study investigating this showed that a combined Rutherrin-PDT increased CD8 + T-cells in a rat glioma model [83]. The immune-modulating effect on the TME would therefore convert the initially cold tumour to a hot tumour that would have higher susceptibility to immunological treatments. The identification of the exact immunological response to SDT and subsequent effects of this on the TME is therefore needed to further assess the clinical role of SDT in cancer treatment both as an individual and combined therapy [84, 85].


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